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B2, presents chemotactic and Gram-negative selectivebactericidal activities Constance Auvynet1,2,*, Pierre Joanne2,, Julie Bourdais3, Pierre Nicolas2,, Claire Lacombe2,4, and Yvonne Rose

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B2, presents chemotactic and Gram-negative selective

bactericidal activities

Constance Auvynet1,2,*, Pierre Joanne2,, Julie Bourdais3, Pierre Nicolas2,, Claire Lacombe2,4, and Yvonne Rosenstein1

1 Departamento de Medicina Molecular y Bioprocesos, Instituto de Biotecnologia, Universidad Nacional Auto´noma de Me´xico, Col Chamilpa, Cuernavaca, Morelos, Mexico

2 FRE 2852, Peptidome de la peau des amphibiens, CNRS ⁄ Universite´ Paris–Pierre et Marie Curie, Paris, France

3 Independent scholar, Cuernavaca, Morelos, Mexico

4 UFR Sciences et Technologie, Universite´ Paris 12–Val de Marne, Cre´teil, France

Keywords

antimicrobial peptide; chemotaxis;

dermaseptin; frog skin; peptide–membrane

interactions

Correspondence

Y Rosenstein, Departamento de Medicina

Molecular y Bioprocesos, Instituto de

Biotecnologia, Universidad Nacional

Auto´noma de Me´xico, Avenida Universidad

2001, Col Chamilpa, Cuernavaca, Morelos

62270, Mexico

Fax: +52 73172388

Tel: +52 5 55 66 22 76 63

E-mail: yvonne@ibt.unam.mx

C Lacombe, Laboratoire des Biomole´cules,

Universite´ Pierre et Marie Curie-CNRS-ENS,

4 Place Jussieu, 75252 Paris cedex 05,

France

Fax: +33 1 44 27 55 64

Tel: +33 1 44 27 51 59

E-mail: claire.lacombe@upmc.fr

Present addresses

*INSERM UMR-S 945 Immunite´ et

Infec-tion, Universite´ Pierre et Marie Curie, Paris,

France

Biogene`se des Signaux Peptidiques

(BIOSIPE), ER3-UPMC, Universite´ Pierre

et Marie Curie, Paris, France

Antimicrobial peptides participate in innate host defense by directly elimi-nating pathogens as a result of their ability to damage the microbial mem-brane and by providing danger signals that will recruit innate immune cells

to the site of infection Dermaseptin DA4 (DRS-DA4), a new antimicrobial peptide of the dermaseptin superfamily, was identified based on its chemo-tactic properties, contrasting with the currently used microbicidal proper-ties assessment The peptide was isolated and purified by size exclusion HPLC and RP-HPLC from the skin of the Mexican frog,

Pachymedu-sa dacnicolor MS and amino acid sequence analyses were consistent with the structure GMWSKIKNAGKAAKAAAKAAGKAALGAVSEAM CD experiments showed that, unlike most antimicrobial peptides of the derm-aseptin superfamily, DRS-DA4 is not structured in the presence of zwitteri-onic lipids DRS-DA4 is a potent chemoattractant for human leukocytes and is devoid of hemolytic activity; in addition, bactericidal tests and mem-brane perturbation assays on model memmem-branes and on Escherichia coli and Staphylococcus aureus strains have shown that the antibacterial effects of DRS-DA4 and permeabilization of the inner membrane are exclusively selective for Gram-negative bacteria Interestingly, despite high sequence homology with dermaseptin S4, dermaseptin B2 was not able to induce directional migration of leukocytes, and displayed a broader bactericidal spectrum A detailed structure–function analysis of closely related peptides with different capabilities, such as DRS-DA4 and dermaseptin B2, is criti-cal for the design of new molecules with specific attributes to modulate immunity and/or act as microbicidal agents

Abbreviations

DDK, dermadistinctin K; DiSC3(5), 3,3¢-dipropylthiadicarbocyanine iodide; DMPC, 1,2-dimyristoyl-sn-glycero-3-phosphatidylcholine; DMPG, 1,2-dimyristoyl-sn-glycero-3-phosphatidylglycerol; DRS-B2, dermaseptin B2; DRS-DA3, dermaseptin DA3; DRS-DA4, dermaseptin DA4; DRS-L1, dermaseptin L1; DRS-S1, dermaseptin S1; DRS-S9, dermaseptin S9; DSC, differential scanning calorimetry; ERK, extracellular signal-regulated kinase; fMLP, formyl-methionyl-leucyl-phenylalanine; FPR, formyl peptide receptor; FPRL-1, formyl peptide receptor-like 1; Gal-ONp, 2-nitrophenyl b- D -galactopyranoside; GPCR, G-protein-coupled receptor; ITC, isothermal titration calorimetry; LUV, large unilamellar vesicle; MAPK, mitogen-activated protein kinase; MIC, minimum inhibitory concentration; MLV, multilamellar liposome vesicle; PMN, polymorphonuclear; PTX, pertussis toxin; SDF1-a, stromal cell-derived factor 1a; TFA, trifluoroacetic acid.

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At the interface of innate and adaptive immunity,

antimicrobial peptides have been shown to enhance the

overall immune response [1] The majority of these

peptides are cationic, with a net charge of +2 to +7,

and contain up to 50% hydrophobic amino acids This

amphipathic design, consisting of spatially separated

hydrophobic and charged regions, is believed to allow

the insertion of the peptide into microbial membranes

Until recently, direct antimicrobial activity against

bacteria, fungi, parasites and viruses was considered to

be the primary function of antimicrobial peptides

However, there is now increasing evidence that

anti-microbial peptides are multifunctional molecules of

fundamental importance in host defense, modulating

the innate and adaptive immune systems In addition

to microbicidal activity, a large number of

antimicro-bial peptides, such as the human cathelicidin LL-37

and the defensins, have been found to modulate the

immune response by directing the migration of immune

cells to the site of injury, as well as by activating

leuko-cytes and promoting cytokine release, wound repair,

angiogenesis, and neutralization of microbial products

[2,3] In particular, the chemotactic activity of

antimi-crobial peptides is mostly mediated through

G-protein-coupled receptors (GPCRs) such as the CC-chemokine

receptor 6, the formyl peptide receptor (FPR), and the

formyl peptide receptor-like 1 (FPRL-1) [4–7]

Frog skin is a rich source of antimicrobial peptides,

with more than half of the peptides described to date

having been isolated from South American Hylidae or

European, Asian or North American Ranidae; the

peptides are involved in the defense of the frog against

predation or invading microorganisms More than 80

antimicrobial peptides have been isolated from only 12

species of the Phyllomedusinae subfamily, belonging to

the genera Agalychnis, Hylomantis, Pachymedusa, and

Phyllomedusa Among them, the dermaseptins, a

super-family of structurally and functionally related peptides

produced by the Hylidae family, have potent

micro-bicidal activity at micromolar concentrations against a

wide range of microorganisms (Gram-positive and

Gram-negative bacteria, fungi, yeasts, and protozoa),

but no or little hemolytic activity [8] The microbicidal activity of these lysine-rich linear polycationic peptides, most of which are composed of 24–34 amino acids structured as an amphipathic a-helix in polar solvents,

is thought to result from the interaction of the amphi-pathic a-helical structure with the membrane bilayer of target microorganisms

Most peptides belonging to the dermaseptin super-family have been identified primarily on the basis of their antimicrobial activity However, additional bio-logical functions have been recognized that may, or may not, be directly associated with pathogen clearance For instance, adenoregulin [dermaseptin B2 (DRS-B2)] was first identified as a peptide able to stimulate binding of agonists to A1-adenosine receptors [9], and was further shown to enhance the binding potency of several GPCR agonists [10] Frog skin insulintropic peptide (FSIP), also a member of this superfamily, significantly stimu-lates insulin release in glucose-responsive BRIN-BD 11 cells [11], and dermaseptin S1 (DRS-S1) has been reported to stimulate the microbicidal activity of poly-morphonuclear (PMN) leukocytes [12] and dermaseptin S9 (DRS-S9) to chemoattract PMN leukocytes [13]

We report herein the isolation and characterization

of a new dermaseptin-related peptide, GMWSKIKNA GKAAKAAAKAAGKAALGAVSEAM, named aseptin DA4 (DRS-DA4), according to the new derm-aseptin nomenclature [14] DRS-DA4 was obtained by fractioning the skin exudate of Pachymedusa

dacnicol-or, and was first identified on the basis of its chemo-tactic properties rather than on the classic assessment

of its antimicrobial activity Interestingly, although DRS-DA4 was found to share strong sequence homol-ogy with DRS-B2, it has distinct biological activities DRS-DA4, but not DRS-B2, induced human leukocyte migration and activation mainly through a GPCR, probably FPRL-1; moreover, it was devoid of hemo-lytic activity Unlike DRS-B2, which is active on Gram-positive as well as on Gram-negative bacteria, DRS-DA4 only exhibited direct antibacterial activity

on Gram-negative bacteria, together with perturbation

of the inner membrane against of Gram-negative

Laboratoire des Biomole´cules, Universite´

Pierre et Marie Curie-CNRS-ENS, Paris

cedex 05, France

(Received 12 August 2009, accepted 21

September 2009)

doi:10.1111/j.1742-4658.2009.07392.x

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bacteria The identification of a novel antimicrobial

peptide on the basis of its immunomodulatory capacity

broadens the panel of activities of these peptides

within Hylidae frog genera In addition, our analysis

of the biophysical characteristics and properties of

these peptides contributes to our current knowledge

regarding the different activities of antimicrobial

peptides, and opens the possibility of using them as

templates for the design of new molecules with specific

attributes to modulate immunity and⁄ or act as

micro-bicidal agents

Results

Isolation, purification and structure of DRS-DA4

DRS-DA4 was purified to homogeneity from P

dacni-colorskin exudates by a two-step protocol Specifically,

1.1 mL of skin secretions recovered by gently

squeez-ing the parotoid glands of a ssqueez-ingle livsqueez-ing frog was first

fractionated on a Sephadex G-50 column (Fig 1A),

and each fraction was tested for chemotactic activity

The chemotactic fraction III was further purified by

RP-HPLC on a semipreparative column (Fig 1B) A peak with a retention time of 38.7 min was found to have strong chemotactic activity, inducing the direc-tional migration of human leukocytes (Fig 1B, insert) The sequence of the purified fraction determined by tandem MS (experimental monoisotopic mass of the protonated peptide: 3063.26) and Edman sequencing gave unequivocally the sequence GMWSKIKNAGKA AKAAAKAAGKAALGAVSEAM According to the new dermaseptin nomenclature [14], this peptide was named DRS-DA4

To confirm the sequence and to demonstrate that the biological activities of the purified natural peptide reflected its intrinsic properties, DRS-DA4 was synthe-sized by the solid-phase method After HPLC purifica-tion on a semipreparative column, synthetic DRS-DA4 was indistinguishable from the natural product, eluting exactly at the same position (38.7 min) as the natural corresponding product and giving the same monoi-sotopic mass to charge ratio (3063.28) by MALDI-TOF

MS (data not shown) Further characterization of the conformational and biological properties was performed with the synthetic peptide

A

B

0.0 0.1 0.2 0.3 0.4 0.5 0.6 0.7 0.8 0.9

Fraction III Fraction II

Fraction I

V0

Fraction number

Fig 1 (A) Profile of fractionation of the

P dacnicolor skin exudates on a Sephadex

G-50 column The absorbance at 280 nm is

represented as a solid line (B) RP-HPLC

separation of the recovered fraction III using

a semipreparative column Elution was

achieved with a 0–60% linear gradient of

solvent (dotted line) The arrow points to

the elution position of synthetic DRS-DA4

under the same conditions The absorbance

at 220 nm is represented as a solid line.

Insert: neutrophil migration induced in

response to the peak indicated by the

arrow The peak solution was diluted 1 ⁄ 10

from column 6 to column 1 RPMI medium

was used as negative control (column 0)

and fMLP (100 n M ) as positive control.

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A protein database was screened for similar

sequences, using the embl-ebi fasta 3 program, and

this revealed that DRS-DA4 belonged to the

derm-aseptin superfamily A sequence alignment showed

that the antimicrobial peptides dermaseptin DA3 [DRS-DA3 (PD-33)] [15], DRS-B2 [9], dermadistinctin

K (DDK) [16] and ARP-AC1 [17], isolated from

P dacnicolor, Phyllomedusa bicolor, Phyllomedusa

A

B

Fig 2 (A) Amino acid sequences of DRS-DA4 and DRS-B2, together with some antimicrobial peptides with nearest sequences Identical amino acids between these peptides are underlined (B) Helical wheel projection of the DRS-DA4, DRS-B2, DDK, and DRS-L1 Hexagonal and round backgrounds refer to basic and acid amino acids respectively, pentagonal backgrounds to hydrophilic residues, and squares to hydrophobic residues.

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distincta, and Agalychnis callidryas, respectively,

presented the highest sequence homology with

DRS-DA4 (87.9% identity and 93.9% similarity for

DRS-DA3, 84.8% identity and 97% similarity

for DRS-B2, and 84.8% identity and 93.9% similarity

for DDK; Fig 2A) Both, DRS-DA3 and DRS-B2

have six positive charges (Lys), and only one negative

charge (Glu) The resulting total overall net charge for

DRS-DA4 is +5, whereas DRS-B2 has an overall net

charge of +4, owing to the carboxyamidated end,

three negative charges (Glu) and the extra positive

charge of the N-terminus Figure 2A shows dermaseptin

L1 (DRS-L1) [18] from the lemur leaf frog

Hyloman-tis lemur, which showed only 48.6% identity and

22.4% similarity but whose biological properties led us

to make a comparison The helical wheel projection of

Edmundson showed the partial amphipathic character

of the helix obtained for DRS-DA4, with hydrophobic

residues on one face of the helix and polar or charged

residues on the opposite face, corresponding to a

hydrophobic sector that subtends a radial angle of

160 (Fig 2B)

DRS-DA4 induces directional migration of human

leukocytes

Like natural DRS-DA4, synthetic DRS-DA4 induced

the migration of human neutrophils (Fig 3A) and

monocytes (Fig 3B), with a typical bell-shaped dose–

response curve Maximum activity was observed at a

concentration of 10 lm for both cell types Addition

of various concentrations of DRS-DA4 to the upper

wells of the Boyden chamber abolished the migration

of the cells, suggesting that DRS-DA4 induced

che-motactic movement rather than enhanced random

movement (Fig 3A) As described previously [13],

DRS-B2 did not induce the migration of leukocytes

within the range of concentrations tested for

DRS-DA4

Like formyl-methionyl-leucyl-phenylalanine (fMLP),

most chemotactic antimicrobial peptides induce cell

migration through a seven-helix transmembrane

Gia-protein-coupled receptor such as FPR or FPRL-1

[19] Pretreatment with pertussis toxin (PTX) – a specific

inhibitor of Gia-protein-coupled receptors – prior to

the onset of the chemotactic assay partially inhibited

the motility of neutrophils induced by DRS-DA4 As

expected, neutrophils preincubated with PTX before

exposure to fMLP failed to migrate (Fig 4A)

Preincubation of neutrophils for 30 min with 100 nm

fMLP, an FPRL-1 agonist, inhibited by up to 50% the

migration of the cells in response to a gradient of

DRS-DA4 or fMLP (100 nm) However, preincubation

of the cells with 1.14 nm fMLP, a concentration at which fMLP is considered to be an FPR agonist, did not inhibit the motility of the cells in response to DRS-DA4 or fMLP (100 nm) As expected, preincuba-tion of neutrophils with fMLP (1.14 nm) prior to the chemotaxis assay inhibited the migration induced by fMLP (1.14 nm) (Fig 4A)

Stimulation of a GPCR by chemotactic agonist ligands can lead to the activation of mitogen-activated protein kinase (MAPK) pathways in target cells [20] Incubation of neutrophils with DRS-DA4 (10 lm) or fMLP (100 nm) for 5 or 10 min induced the phos-phorylation of the extracellular signal-regulated kinase (ERK)1⁄ 2 MAPK, but not that of the p38 MAPK (Fig 4B) Preincubation with PTX or PD98059 (a specific inhibitor of MEK1 and MEK2) before the addition of DRS-DA4 (10 lm) or fMLP (100 nm) inhibited the phosphorylation of ERK1⁄ 2 but induced

A

B

Fig 3 DRS-DA4 is a potent chemoattractant of leukocytes (A) Neutrophil migration induced in response to DRS-DA4 or fMLP (100 n M ) as a positive control and medium as a negative control Addition of the same concentrations of DRS-DA4 to the upper and lower wells of the chemotaxis chamber abolished the chemotactic effect (B) Monocyte migration in response to DRS-DA4, DRS-B2 or SDF1-a (1 n M ) as positive control The data shown represent the average cell migration of triplicate wells Similar results were obtained from three different experiments.

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the phosphorylation of p38 Moreover, pretreatment

of neutrophils with PD98059 led to decreased

migra-tion in response to DRS-DA4 or fMLP (100 nm),

whereas SB202190, a specific inhibitor of the p38

MAPK, did not (Fig 4C) All together, these results

suggest that DRS-DA4 chemotactic activity is medi-ated through a GPCR, probably FPRL-1, and that it

is coupled to the ERK1⁄ 2 MAPK pathway

DRS-DA4, but not DRS-B2, is only microbicidal for Gram-negative bacteria

DRS-DA4 exhibited good to moderate antimicrobial activity against all of the Gram-negative strains tested,

as no colony was counted when the peptide–Escherichia coli (both E coli strains tested) or peptide–Pseudo-monas aeruginosa mixtures were incubated on LB agar plates overnight, indicative of a bactericidal effect How-ever, no microbicidal activity was detected against any

of the Gram-positive strains tested, at concentrations up

to 100 lm In comparison, DRS-B2 presented strong antimicrobial activities against both Gram-negative and Gram-positive strains (Table 1) No hemolytic or apop-totic activities were observed on rat blood cells or leuko-cytes with DRS-DA4 or DRS-B2, respectively, for concentrations up to 100 lm (data not shown)

DRS-DA4 interacts preferentially with 1,2-dimyristoyl-sn-glycero-3-phosphatidylglycerol (DMPG) vesicles mimicking Gram-negative bacteria When we used the membrane potential-sensitive dye 3,3¢-dipropylthiadicarbocyanine iodide [DiSC3(5)] to assess the ability of the peptide to damage, and thus depolarize, prokaryotic membranes [21,22], we found that addition of DRS-DA4 resulted in concentration-independent increases in DiSC3(5) fluorescence within the range 10–50 lm, indicative of equivalent membrane depolarization for both strains tested, i.e E coli and Staphylococcus epidermidis (Table 1) Thus, in contrast

to its Gram-negative-specific microbicidal activity, DRS-DA4 was able to depolarize the membranes of Gram-positive and Gram-negative bacteria

To further characterize the bactericidal activity

of DRS-DA4, we measured its ability to damage the bacterial membrane Permeabilization of the inner mem-brane can be assessed by monitoring the b-galactosidase substrate 2-nitrophenyl b-d-galactopyranoside (Gal-ONp) In E coli strain ML35p and in Staphylococ-cus aureusstrain ST1036, which lack lac permease, Gal-ONp entry is blocked by the integrity of the inner mem-brane; if Gal-ONp crosses this barrier, it can be cleaved

by the cytoplasmic b-galactosidase, resulting in a color change from clear to yellow, reflecting membrane dam-age As shown in Fig 5A, the time needed to obtain the maximum Gal-ONp hydrolysis in E coli increased as the concentration of DRS-DA4 decreased On the other hand, no permeabilization of S aureus ST1065 was

10 ’ 10 ’ 5 ’ 10 ’ 10 ’ 10 ’ 10 ’ 10 ’ 10 ’ 10 ’

pErk1/2 Erk1/2 pp38 p38

A

B

C

Fig 4 DRS-DA4-induced neutrophil migration through a

seven-transmembrane GPCR (A) DRS-DA4-induced chemotaxis is

medi-ated through a GPCR Neutrophils were preincubmedi-ated with medium,

PTX, fMLP (1.14 n M ), or fMLP (100 n M ) prior to challenge with

DRS-DA4, fMLP (1.14 n M ) or fMLP (100 n M ) (B) ERK1 ⁄ 2

phosphor-ylation is induced in response to DRS-DA4 Human neutrophils

were treated with medium, DRS-DA4, or fMLP, with or without

preincubation with PTX and ⁄ or PD98059 The same membrane

was stripped and blotted with antibody against ERK1 ⁄ 2 or antibody

against p38 Similar results were obtained from three separate

experiments (C) ERK1 ⁄ 2 phosphorylation is necessary for

DRS-DA4-induced Human neutrophils were preincubated with PD98058

(ERK1⁄ 2 inhibitor) or SB202190 (p38 inhibitor) prior to the

chemo-taxis assay, as described in (A) The data shown are representative

of two independent experiments.

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observed for concentrations of DRS-DA4 up to 100 lm

(Fig 5B and data not shown) These results indicate

that the bacterial membrane of Gram-negative strains is

one of the main targets of DRS-DA4

Differential scanning calorimetry (DSC) is a

power-ful, nondisturbing thermodynamic technique that is

useful for the study of lipid–protein interactions in

model membranes as well as for the evaluation of

anti-microbial peptide interactions with lipid bilayer model

membranes [23] We evaluated the effect of various

concentrations of DRS-DA4 on the pretransition and

the main transition of the zwitterionic

1,2-dimyristoyl-sn-glycero-3-phosphatidylcholine (DMPC) and anionic

DMPG membranes used as models for eukaryotic and

prokaryotic plasma membranes, respectively DSC

thermograms illustrating the effect of the incorporation

of increasing quantities of DRS-DA4 on the

thermo-tropic phase behavior of multilamellar liposome

vesi-cles (MLVs) of DMPG or DMPC are shown in

Fig 5C,D In the absence of the peptide, DMPG

exhibited two endothermic events: a less energetic

pre-transition near 12.8C, arising from the conversion of

the lamellar phase to the rippled gel phase, and a

sec-ond, more energetic, main transition at 23.2C,

result-ing from the conversion of the rippled gel phase to the

lamellar liquid-crystalline phase These results, together

with the enthalpy values for the pretransition

( 1 kcalÆmol)1) and the main transition (8.8 kcalÆ

mol)1), are comparable with previous data [24,25] The

incorporation of DRS-DA4 into DMPG MLVs

signifi-cantly altered their thermotropic phase behavior The

presence of the peptide abolished the pretransition,

even at the lowest concentration tested (peptide/lipid

ratio 1 : 100), which is within the same range of order

as the minimum inhibitory concentration (MIC) for

Gram-negative strains Increasing concentrations of

DRS-DA4 broadened the DMPG main phase

transi-tion peak, probably because of a loss of cooperativity

during the lipid fusion resulting from the insertion of the peptide molecules The main transition was totally abolished at 1 : 20 peptide⁄ lipid ratios, indicating total disorganization of lipid DMPG bilayer (Fig 5C) Like those of DMPG, aqueous dispersions of DMPC showed two endothermic transitions, a pretran-sition occurring at 13.1C with an enthalpy of about 1.1 kcalÆmol)1, and a main transition at 23.9C with

an enthalpy of 11 kcalÆmol)1, which was again within the range previously published [24,25] At low con-centrations of DRS-DA4 (1 : 100 peptide⁄ lipid ratio),

no modifications of the thermogram were observed The pretransition was then gradually abolished as the peptide/lipid ratio increased At greater ratios only (1 : 20 peptide⁄ lipid ratio), DRS-DA4 induced some changes to the DMPC melting profile, indicating an interaction with this type of vesicle, whereas the main transition peak was still present, without modification

of its temperature

CD spectra of DRS-DA4 (Fig 5E) revealed that DRS-DA4 adopted an a-helix conformation in the presence of DMPG vesicles, whereas it remained as a random coil in aqueous solution and in the presence of DMPC liposomes, whatever the peptide/lipid ratio (data not shown) In the presence of DMPG vesicles, the spectrum of DRS-DA4 showed a profile with minima at 208 and 220 nm, suggesting a major contri-bution of a-helix ( 40%) The conformational propensity of DA4 contrasts with that of DRS-B2, which has been found to be structured as an a-helix (55%), whatever the lipidic composition of the vesicles [26]

Discussion

Hundreds of antimicrobial peptides have been isolated from frogs, but very few studies dealing with their immunomodulatory capacities have been published

Table 1 Antimicrobial activities of DRS-DA4 and DRS-B2 Bacterial strains were considered to be resistant (R) when their growth was not inhibited by peptide concentrations up to 100 l M The data shown correspond to the MIC (l M ) E coli 363 ATCC 1175 or S epidermidis BM

3302 transmembrane potential changes were induced by DRS-DA4 and assessed with the DiSC3(5) probe: membrane depolarization was monitored by an increase in fluorescence after the addition of peptide at the MIC Triton X-100 was used to fully collapse the membrane potential B, bactericidal; ND, not determined.

a

See ref [26].

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The first antimicrobial peptide shown to exhibit

immu-nological properties was DRS-S1, a 34 amino acid

cationic antimicrobial peptide, which stimulates the

microbicidal activity of PMN leukocytes [12] A few

other frog peptides, temporin A, rana-6 and pLR-like

peptides and, recently, another dermaseptin-related peptide, DRS-S9, have been shown to be microbicidal

as well as immunomodulatory [13,27–29] Here, we report the isolation, by screening for chemotactic activ-ity, of a new member of the dermaseptin superfamily,

0.0 0.5 1.0 1.5 2.0

0 2000 4000 6000 8000 10 000 12 000 14 000

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Time (s)

0 2000 4000 6000 8000 10 000 12 000

Time (s)

20 µ M

10 µ M

5 µ M 1.2 µ M 0.3 µ M Control

5 10 15 20 25 30 35 40

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Temperature (°C)

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190 200 210 220 230 240 250 260 –6

–4 –2

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Wavelength (nm)

DMPG DMPC Buffer

Fig 5 The bactericidal capacity of DRS-DA4 is linked to its interaction with DMPG (A, B) Kinetics of bacterial membrane leakage of E coli ML35p (A) and S aureus ST1065 (B) after treatment with increasing concentrations of DRS-DA4 The membrane leakage was followed by measuring Gal-ONp hydrolysis at 405 nm (C, D) DSC heating thermograms illustrating the effects of DRS-DA4 on the thermotropic phase behavior of DMPG (C) and DMPC (D) MLVs The top scan corresponds to the lipid alone, and the peptide ⁄ lipid molar ratios of the lower scans are indicated Thermodynamic parameters are given in Table 2 (E) CD spectra of DRS-DA4 in buffer, DMPG and DMPC with a peptide ⁄ lipid molar ratio of 1 : 50 The data shown are representative of three experiments.

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DRS-DA4, from the defensive skin secretion of the

Mexican leaf frog P dacnicolor

We showed that DRS-DA4 triggered the in vitro

directional migration of human neutrophils and

mono-cytes with a typical bell-shaped dose–response curve,

and a maximal response at a concentration of 10 lm

Our results suggested that the chemotactic effect was

mediated by the low-affinity Gia-protein-coupled

recep-tor FPRL-1, recently reported to interact with many

different ligands, such as fMLP, the a-helical

antimi-crobial peptides LL-37 and temporin A, and the

amy-loidogenic peptides Ab1–42 and, presumably, DRS-S9

[13,27,30,31] (Fig 4B) Whether FPRL-1 interacts with

its various agonists through different functional

domains remains to be investigated In addition, as

only human leukocytes were used in this study, it

remains to be discovered whether DRS-DA4 is also

capable of recruiting frog leukocytes Indeed, frog

pep-tides are secreted at the outer surface of the skin, and

it is presently not known whether they also enter the

blood circulation or inner tissues to act on leukocytes

or to modulate other biological functions

As described for other agonists [20], the interaction

of DRS-DA4, presumably with FPRL-1, led to the

activation of the ERK1⁄ 2 MAPK pathway, but not to

that of the p38 MAPK pathway The chemotaxis data

suggested that ERK phosphorylation, but not that of

p38, was necessary for the chemotactic process

Inter-estingly, inhibition of FPRL-1 by PTX has been

reported to activate the p38 pathway [32] Consistent

with this, inhibition of ERK1⁄ 2 phosphorylation by a

specific inhibitor (PD98059) activated the p38 MAPK

pathway in response to DRS-DA4, suggesting that

DRS-DA4 may additionally bind to a non-GPCR

receptor and, through a p38-dependent pathway,

con-trol other cell functions such as degranulation or

cyto-kine⁄ chemokine gene expression and release This is

in accordance with studies indicating that inhibition

of one signaling pathway could activate another one

[27,33]

The 32 amino acid DRS-DA4 exhibits the typical

characteristics of the dermaseptin superfamily: it is a

linear, Lys-rich cationic peptide with the conserved

Trp at position 3 A blast search revealed high

sequence homology of DRS-DA4 with DRS-DA3

(PD-33), also isolated from P dacnicolor, and, more

surprisingly, with DRS-B2 isolated from Ph bicolor,

DDK from Ph distincta, and ARP-AC1 from A

cal-lidryas[17] (Fig 2A) However, unlike these other

pep-tides, DRS-DA4 is not carboxyamidated Modeling

antimicrobial peptides as idealized helices revealed

their highly amphipathic nature, with hydrophobic

residues on one face of the helix and polar or charged

residues on the opposite face, leading us to propose that the amphipathic a-helix structure is an important feature of these membrane-permeating peptides Secon-dary structure prediction methods and CD spectros-copy have also shown that dermaseptins contain 45–90% helix in structure-promoting solvents [16,26, 34,39] We illustrated here by CD (Fig 5E) that DRS-DA4 was randomly coiled in water and, unexpectedly, also in the presence of DMPC vesicles In contrast, DRS-DA4 adopted an a-helical structure in the pres-ence of DMPG vesicles, consistent with its cidal action against prokaryotic cells Thus, DRS-DA4 fits the model proposed by Khandelia et al [35], in which the composition of the target membrane (zwitterionic or anionic) modulates the extent of helical content induced in antimicrobial peptides CD results are con-sistent with data from the calorimetric tests and iso-thermal titration calorimetry (ITC) experiments with DMPC large unilamellar vesicles (LUVs) (data not shown), again suggesting very weak or no interaction with this lipid In contrast, strong perturbations of the pretransition and the main phase transition (Fig 5C and Table 2) were recorded in the presence of DMPG, suggesting that electrostatic interactions participate in the peptide–lipid interaction, and that DRS-DA4 is able to penetrate the acyl chain region

A highly cationic charge such as that of DRS-DA4 favors the accumulation of peptides on negatively charged DMPG bilayers via electrostatic interactions, suggesting that the bactericidal activity of DRS-DA4 towards Gram-negative bacteria results from the pref-erential binding of the peptide to the negatively charged lipopolysaccharides of the outer membrane, and that the subsequent membrane damage occurs through hydrophobic interactions with the inner target membrane, which is rich in neutral phosphatidyletha-nolamine This hypothesis is supported by a study showing that helical amphipathicity prevails over hydrophobicity in interfacial binding, underlining the

Table 2 Thermodynamic parameters obtained by DSC for the interaction of DRS-DA4 with MLVs of either DMPG or DMPC –, no pretransition observed.

Lipid

Peptide ⁄ lipid ratio

Pretransition Transition

T (C)

DH (kcalÆmol)1) T (C)

DH (kcalÆmol)1)

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importance of amphipathicity as a driving force

for cell lytic activity In addition, conformational

constraints and appropriate positioning of aromatic

residues for the formation of hydrophobic clusters

have been shown to be critical for antimicrobial

activ-ity and selectivactiv-ity [36] Moreover, the presence of

regions with different order and polarity within the

membrane has shown the existence of domains

enriched in phosphatidylethanolamine or

phosphatidyl-glycerol, localized in highly curved regions of the

bacterial membrane [37] Segregation of the membrane

lipid components, leading to clustering of anionic

lip-ids through an induced lateral-phase separation, and

the subsequent perturbation of existing domains of the

membrane has been proposed as a mechanism

contrib-uting to the antimicrobial activity of numerous

antimi-crobial peptides In agreement with this, as they have

significant amounts of both anionic and zwitterionic

lipids, most Gram-negative bacteria are more

suscepti-ble to this membrane-disrupting mechanism

Consis-tent with this, and as reported for oligo-acyl lysine

[38], we found that Gram-negative bacteria were killed

by DRS-DA4

It has been suggested that an uninterrupted section

of five hydrophobic residues, as identified on the

heli-cal wheel, is sufficient for good antimicrobial activity,

with reduced hemolysis [39] This is the case for

DRS-DA4 and DDK However, despite strong

sequence homology with DRS-B2, DRS-DA4 exhibits

distinct biological activities Contrasting with the wide

microbicidal spectrum of DRS-B2, DRS-DA4’s

ricidal capacity was selective for Gram-negative

bacte-ria (Table 1), and, whereas DRS-B2 did not induce

leukocyte motility, DRS-DA4, at equivalent

concentra-tions, was a potent chemotactic agent for PMN

leuko-cytes (Fig 4B) A comparison of the helical wheel

projections of DRS-B2, DDK, ARP-AC1 and DRS-L1

(a dermaseptin with a similar selectivity for

Gram-negative bacteria [18]) revealed that, if the last three

residues – which are rarely involved in the a-helix

formation – are disregarded, these peptides exhibit an

amphipathic distribution (Fig 2B) The main

informa-tion provided by the Edmundson projecinforma-tion is that

DRS-DA4 and DRS-L1, the two peptides that are

active only on Gram-negative strains, do not expose a

negative residue on the apolar face, as do DRS-B2 and

DDK, which have a Glu at position 31, or ARP-AC1,

with an Asp at position 27 In agreement with this, the

position of acidic residues seems to be a critical

para-meter for the antibacterial activity against

Staphylo-coccusstrains [40] However, the ability of a peptide to

depolarize the cytoplasmic membrane does not

neces-sarily correlate with bactericidal activity Indeed, our

data show that, like plasticins, DRS-DA4 was able to depolarize the membrane of Gram-positive (S epider-midis ST1065) as well as of Gram-negative (E coli ML35p) strains of bacteria, but it was toxic only for Gram-negative strains (Table 1)

In contrast to the situation with clinically used anti-biotics, resistance to natural antimicrobial peptides is not frequent, raising interest in the use of antimicro-bial peptides to fight antibiotic-resistant microbes Moreover, recent data have suggested that antimicro-bial peptides participate actively in preventing the appearance of resistant mutants, and are thus the last line of defense dealing with persistent infections [41] Our data showing the antibacterial potency of DRS-DA4, resulting from its ability to interact with anionic model membranes and to induce directional locomotion of mammalian cells through a receptor, presumably FPLR-1, highlight the multifunctionality

of antimicrobial peptides as antibiotics and immuno-modulatory molecules [3,42,43] Modification of DRS-DA4 to enhance its direct bactericidal effect and to extend its antibacterial activity to Gram-positive strains, without compromising its immunomodulatory potency, could be achieved, as for dermaseptin S4, through acylation [44], or, as for magainin 2 analogs, amidation [45] This new class of peptides will be use-ful for therapeutic application purposes

Experimental procedures

Frogs

Male and female specimens of P dacnicolor were captured

in the state of Morelos (Mexico) and housed in a nonsterile environment, in a large plastic container covered by a fence phyllodendron, potos and dracena were used as perches, and a water bowl was provided for nocturnal baths Once a week, the frogs were fed with crickets

Purification of the peptide

Fresh skin exudate was recovered by gently squeezing the latero-dorsal portion of a frog skin, resuspended in de-ion-ized water, and centrifuged for 15 min at 400 g The super-natant was first fractionated by size exclusion

(60· 0.75 cm) eluted with 10% acetic acid Absorbance was monitored at 280 nm Three main fractions were obtained and tested for their chemotactic activity Fraction III was further fractionated by RP-HPLC on a semipreparative col-umn (Nucleosil 5 lm C18, 250· 10 mm), using a solvent system composed of water containing 0.1% trifluoroacetic acid (TFA) as solvent A, and acetonitrile containing 0.07%

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