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Hip and knee flexion contractures occur when patients compensate for loss of sagittal balance by assuming a crouched position.. Deformity resulting from inflammation and fusion of the sacr

Trang 1

Erik N Kubiak, MD, Ronald Moskovich, MD, Thomas J Errico, MD, and Paul E Di Cesare, MD

Abstract

Ankylosing spondylosis (AS) is a

se-ronegative inflammatory disease of

unknown etiology characterized by

inflammation in the axial skeleton

The sacroiliac joints are often

in-volved initially, followed by

enthe-sopathy in the paravertebral

zyga-pophyseal joints and disk spaces

Enthesopathy is inflammation at the

site of tendon insertions that

predis-poses to the development of fibrosis

and calcification; left untreated, it

leads to fusion of the zygapophyseal

joints and intervertebral disk space

This fusion in turn can lead to a

fixed, hyperkyphotic posture and

compromised sagittal balance Hip

and knee flexion contractures occur

when patients compensate for loss

of sagittal balance by assuming a

crouched position Degeneration

and ankylosis of peripheral joints,

such as the hip, knee, and shoulder,

may ensue Deformity resulting from inflammation and fusion of the sacroiliac joints, spine, and hips leads to severe functional impair-ment in approximately 30% of pa-tients.1Several diagnostic and treat-ment strategies are available for early and late manifestations of AS

Epidemiology

AS typically affects young adults, most commonly males (M:F = 3:1) in their second through fourth de-cades The incidence of AS in North America is reported to be 1 to 2 per 1,000.2Worldwide prevalence is up

to 0.9%.3Fifteen percent to 20% of patients with AS have a positive family history of the disease AS

is linked to HLA-B27; 80% to 95%

of patients with AS are

HLA-B27–positive.2Carriers of this gene have a 16% to 50% increased risk of developing AS, but other genetic factors are likely involved as well.4 HLA-B27 is present among the in-habitants of Eurasia, North Africa, and North America; it is virtually absent among the aboriginal pop-ulations of Australia and South America.2 Although both HLA-B27–negative and HLA-B27–posi-tive AS patients have similar artic-ular manifestations, the former usually develop the disease at an older age and lack a positive family history AS tends to occur at a later age in females than in males, and di-agnosis is often delayed.2

Dr Kubiak is Resident, Musculoskeletal Research Center, NYU–Hospital for Joint Diseases Depart-ment of Orthopaedic Surgery, New York, NY Dr Moskovich is Associate Chief, Spine Service, Mus-culoskeletal Research Center, NYU–Hospital for Joint Diseases Department of Orthopaedic Sur-gery Dr Errico is Chief, Spine Service, Muscu-loskeletal Research Center, NYU–Hospital for Joint Diseases Department of Orthopaedic Sur-gery Dr Di Cesare is Chief, Adult Reconstruc-tive Service, Musculoskeletal Research Center, NYU–Hospital for Joint Diseases Department of Orthopaedic Surgery.

None of the following authors or the departments with which they are affiliated has received anything

of value from or owns stock in a commercial com-pany or institution related directly or indirectly

to the subject of this article: Dr Kubiak, Dr Mos-kovich, Dr Errico, and Dr Di Cesare Reprint requests: Dr Di Cesare, Musculoskele-tal Research Center, NYU–HospiMusculoskele-tal for Joint Dis-eases, 301 E 17th Street, New York, NY 10003 Copyright 2005 by the American Academy of Orthopaedic Surgeons.

Ankylosing spondylitis is an inflammatory disease of unknown etiology that affects

an estimated 350,000 persons in the United States and 600,000 in Europe,

pri-marily Caucasian males in the second through fourth decades of life Worldwide, the

prevalence is 0.9% Genetic linkage to HLA-B27 has been established Ankylosing

spondylitis primarily affects the axial skeleton and is characterized by inflammation

and fusion of the sacroiliac joints, spine, and hips The resultant deformity leads to

severe functional impairment in approximately 30% of patients Orthopaedic

man-agement primarily involves correction of hip deformity through total hip

arthro-plasty and, less frequently, correction of spinal deformity with spine osteotomy.

Closing wedge osteotomies have the lowest incidence of complications Whether

pa-tients with ankylosing spondylitis are at increased risk for heterotopic ossification

remains controversial, but comparison with age- and sex-matched counterparts

suggests no dramatically higher risk Because of the high rate of missed fractures

and complications after minor trauma in patients with ankylosing spondylitis,

plain radiographs are usually not sufficient for evaluation Thorough patient

as-sessment should include a comprehensive history, physical examination, and

lab-oratory studies.

J Am Acad Orthop Surg 2005;13:267-278

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Not everyone who is HLA-B27–

positive develops AS; it is likely that

other genetic and/or environmental

factors play a role in the etiology.5

One recent theory is that AS is an

autoimmune disorder that occurs

subsequent to a Klebsiella pneumoniae

infection in HLA-B27–positive

indi-viduals, with molecular mimicry

be-tween HLA-B27 and K pneumoniae

bacterial antigens.6 Elevated levels

of antibodies to K pneumoniae have

been noted in AS patients Elevated

IgA levels in AS patients may reflect

mucosal immunity to a persistent

bacterial infection and may be the

autoimmune trigger for AS,

idio-pathic bowel disease, and other

re-active arthritides No causal

rela-tionship has been established

between AS and other autoimmune

diseases, but the incidence of AS in

patients with idiopathic bowel

dis-ease is approximately 3.7%.7

Pathophysiology

The pathognomonic feature of AS is

a combination of inflammation and

bony destruction at the site of

ten-don insertion (ie, enthesopathy)

Pannus or fibroblastoid tissue and

inflammatory cell infiltrates invade

the bone adjacent to entheseal

at-tachments; new bone formation in

response to inflammation leads to

ankylosis of affected joints

Preferen-tial involvement of the

zygapo-physeal joints has been noted on

magnetic resonance imaging (MRI)

Ankylosis and subsequent loss of

motion of the zygapophyseal joints

leads to syndesmophyte formation,

resulting in the characteristic

“bam-boo spine” of individuals with AS

(Fig 1) Enthesopathy also can occur

in peripheral joints, leading to

sy-novitis-like symptoms and joint

de-generation.8Other manifestations of

AS include inflammatory bowel

dis-ease, psoriasis, uveitis/iritis (25%),9

pulmonary fibrosis, aortitis, and genitourinary problems, with prev-alence up to 40%.10,11

Clinical History

Patients usually present with symp-toms and physical findings consistent with the nonmechanical and inflam-matory nature of AS Onset is often insidious; patients typically cannot give the precise time of onset or even pinpoint the initially affected site

These individuals frequently present

in the second through fourth decades

of life with low back pain and stiff-ness, particularly in the morning, at night, or after prolonged periods of sitting and/or recumbency Chest wall pain is a frequent complaint, par-ticularly with deep inhalation Back pain not relieved by recumbency and the persistent discomfort may com-pel the individual to leave the bed at night.12Patients also may have but-tock pain that radiates down the pos-terior thigh Enthesopathy involving the Achilles and plantar tendon inser-tions with associated symptoms also

is common Some patients experience

no pain but present with increasing stiffness in the hips and spine As with other inflammatory diseases, symp-toms often improve with exercise

Physical Examination

A systematic examination of the en-tire patient is imperative Ocular pain, scleral redness, and photophobia are often indicative of acute anterior uve-itis and may be clinically apparent be-fore other symptoms or signs of AS appear Chest expansion measured at the fourth intercostal space typically

is limited to <2.5 cm after fusion of the costovertebral joints; the patient then becomes an obligate diaphrag-matic breather Total lung volume and vital capacity usually are preserved;

the diaphragm excursion is affected only in the patient with severe

tho-racic kyphosis Patients become tachypneic when the degree of ky-phosis compresses the abdominal cavity, thereby limiting

diaphragmat-ic excursion Pulmonary function test-ing should be performed before sur-gery on all patients with AS Aortic and mitral regurgitation murmurs can occur as a consequence of aorti-tis in patients with long-standing dis-ease Right bundle branch block and varying degrees of atrioventricular block occur in up to 30% of patients

as a result of fibrosis extending from the aorta to the intraventricular sep-tum.13

Early in the disease, examination

of the sacroiliac joints may reveal ten-derness to palpation, which can be

ex-Figure 1 Lateral radiograph of the lumbar spine in a 46-year-old man demonstrating the flowing syndesmophytes and posterior ele-ment fusion consistent with bamboo spine Disk spaces are often maintained, especially

in the lumbar spine Anteriorly, osteophytes are often oriented vertically rather than hor-izontally, which is common in spondylitis and diffuse idiopathic skeletal hyperostosis.

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acerbated by hyperextension of the

hips Patients with hip involvement

develop hip flexion contractures

Some patients exhibit loss of

lordot-ic curvature of the lumbar spine and

loss of spine movement in all planes

Patients with decreased mobility of

the thoracolumbar spine have an

ab-normal Schober sign—that is, lack of

at least a 5-cm increase in distance

from a midline point 5 cm below and

10 cm above the posterior superior

il-iac spine, measured in both the erect

standing position and full forward

flexion Late in the disease, the

cer-vical spine can become rigid, with

loss of flexion and extension

Chin-brow angle, occiput-to-wall distance,

and gaze angle are used to evaluate

functional deformity involving the

cervical spine (Fig 2) Most patients

are neurologically normal, with

pres-ervation of deep tendon reflexes A

slow, progressive cauda equina

syn-drome may appear late in the course

of the disease

Assessment of Deformity

Correction of deformity most often

is performed to improve the

pa-tient’s visual field, respiratory

func-tion,14 balance, sitting position,

swallowing function, or ambulation

Determining the site of deformity is

critical before surgically treating the

deformity Flexion contracture of the

hips, loss of lumbar lordosis, and

progressive thoracic and cervical

ky-phosis all may contribute to the

functionally disabling stooped

pos-ture of patients with AS Deformity

must be assessed both clinically and

radiographically as part of

preoper-ative planning The level of

defor-mity will aid in determining the

benefit of total hip arthroplasty

(THA) and/or the level of corrective

spine osteotomy and degree of

cor-rection necessary

The chin-brow angle is the angle

formed by the vertical line drawn

from the chin to the brow with the

pa-tient’s hips and knees fully extended (Fig 2) Occiput-to-wall distance is measured to grossly determine the patient’s sagittal balance The hori-zontal distance from the occiput to the wall is measured with the patient’s buttocks and heels against the wall, with hips and knees extended Clin-ically, occiput-to-wall distance should

be 0 to 2 cm The degree of hip flex-ion contracture is determined by placing the patient supine with the lumbar spine pressed flat to the ex-amining table, then measuring the angle that each femur makes with the horizontal

Normal sagittal balance places the center of mass just in front of the S1 vertebral body Radiographically, a plumb line drawn from the center of the C7 vertebral body should just touch the anterior edge of the body

of S1 on the lateral radiograph Nor-mal individuals are able to compen-sate for small changes in sagittal bal-ance through changes in sacral inclination; hip, knee, and ankle flex-ion; or extension In patients with AS, these normal compensatory changes are ameliorated by loss of motion in these areas.15

Laboratory Evaluation

Baseline laboratory investigation in pa-tients suspected of having AS should include a basic metabolic panel, com-plete blood count, erythrocyte sedi-mentation rate (ESR), C-reactive pro-tein (CRP) level, and rheumatoid factor

Although routine HLA-B27 status is not required to make a diagnosis of

AS, it is helpful in the differential di-agnosis The clinician should be sus-picious of a diagnosis of AS in patients who are HLA-B27–negative Patients

in the active phase of the disease have mildly elevated ESR, CRP level, and white blood cell count In most cases, rheumatoid factor is negative and HLA-B27 is positive Patients with chronic AS may have a normocytic, normochromic anemia

Imaging Studies

The sacroiliac joints usually are the first joints affected in patients with

AS Sacroiliitis initially presents as a widening of the sacroiliac joints with progressive sclerosis of the joint margins, leading to eventual bony fusion across the joints with subsequent loss of sclerosis Care must be taken to disregard congen-ital deformities and degenerative changes as well as osteitis conden-sans ilii (symmetric sclerosis on the iliac sides of both sacroiliac joints without erosions, seen in women who have borne children) Standard anteroposterior radiographs of the pelvis are inadequate to fully eval-uate the sacroiliac joints For accu-rate diagnosis, a Ferguson view of the pelvis (anteroposterior with the x-ray tube tilted 30° cephalad) and oblique radiographs are required Hip involvement presents as ossifi-cation of the ligamentous origins and insertions about the trochanters, iliac crests, and ischial tuberosities Late findings include loss of joint

Figure 2 Measurement of occiput-to-wall distance, gaze angle, and chin-brow angle.

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space, sclerosis, and fusion with loss

of sclerosis at the hips

The ascending osseous changes of

the cervical, thoracic, and lumbar

spine lead to the characteristic

bam-boo spine in advanced stages of the

disease The erosive sclerotic

process-es that occur in the spine are

differ-ent from those that occur in diffuse

idiopathic skeletal hyperostosis

(DISH, also known as Forestier’s

dis-ease) AS can be erroneously

diag-nosed in patients who have

ankylo-sis of the spine for other reasons

Spine lesions in AS are the result of

wedging of vertebral bodies and

os-sification of disk spaces leading to a

rigid kyphotic spine Enthesopathy of

the zygapophyseal joints leads to

fu-sion of the posterior elements The

bone loss and reabsorption that lead

to these deformities occur early in

young persons with active disease.16

Erosions at the discovertebral

junc-tions are called Romanus lesions;

scle-rosis in this region gives rise to

so-called “shining corners.” In AS, the

syndesmophytes are bilateral and

have their insertions at the upper and

lower margins of adjacent vertebrae;

in DISH, the syndesmophytes are

larger and asymmetric, with

nonmar-ginal vertebral insertions consistent

with their status as ligamentous

os-sifications Additionally, DISH spares

the sacroiliac joints and is not

asso-ciated with HLA-B27 In patients with

AS, the zygapophyseal joints are

pre-dominantly involved and frequently obliterated by fusion Four other spondyloarthropathies—psoriatic ar-thritis, reactive arar-thritis, arthritis as-sociated with inflammatory bowel disease, and undifferentiated spondy-loarthropathy—can have

radiograph-ic changes similar to those of AS

Both computed tomography (CT) and MRI are more sensitive than plain radiographs for detecting early dis-ease In a review of imaging tech-niques for sacroiliitis, Braun et al17 ad-vocated the use of CT for evaluation

of erosions and regional ankylosis

MRI is the most sensitive instrument for detecting early disease, with a sen-sitivity of 95% and a specificity of 100%.18Early detection may be im-portant in determining which pa-tients would likely benefit from immune-modulating medication reg-imens when radiographic evidence is lacking Bone scans are unreliable, al-though a negative bone scan should create suspicion of an AS diagnosis

in a patient with sacroiliitis or low back pain

Diagnostic Criteria

The Rome criteria for AS, established

in 1961, were modified by the Amer-ican Academy of Rheumatology in

1966 and renamed the New York clas-sification The New York criteria were modified in 1984 by van der Linden

et al19to improve sensitivity and re-tain specificity (Table 1) The modi-fied New York criteria combine clin-ical and radiographic findings for the diagnosis of AS Radiographic eval-uation (eg, Ferguson’s view, oblique views) of the sacroiliac joints is used

to determine the degree of the sacro-iliitis, which is graded 0 (normal), 1 (suspicious), 2 (minimal sacroiliitis),

3 (moderate sacroiliitis), and 4 (anky-losis) Limitations of this system are

an inability to detect all sacroiliac and/or spinal involvement on plain radiographs, failure to include pa-tients with other forms of spondyloar-thropathy, and an assumption that the duration of inflammatory back pain has exceeded 3 months The presence

of HLA-B27 is not necessary to make the diagnosis

Nonsurgical Management

Nonsteroidal anti-inflammatory drugs (NSAIDs)—specifically, in-domethacin—remain the mainstay of medical therapy Cyclooxygenase-2 inhibitors offer the potential benefits

of decreased gastrointestinal morbid-ity, but their efficacy in the treatment

of AS remains to be proved Sulfasala-zine, methotrexate, thalidomide, and anti–tumor necrosis factor (TNF)-α agents (infliximab, etanercept, adal-imumab) are frequently used in long-term AS management.20 Indometha-cin and radiation therapy have been employed to manage ankylosis and myositis ossificans In 2003, the As-sessment in Ankylosing Spondylitis Working Group recommended that anti–TNF-α be used to treat patients with AS when any of the following exists: (1) a definitive diagnosis of AS; (2) presence of the disease for at least

4 weeks; (3) presence of refractory dis-ease (defined by the failure of two types of NSAIDs during a single 3-month period); (4) failure of local corticosteroid injection into inflamed sacroiliac joints; (5) failure of sul-fasalazine in patients with

peripher-Table 1

The Modified New York Criteria for Diagnosing Ankylosing Spondylitis19

Clinical Criteria

1 Low back pain of at least 3 months’ duration improved by exercise and

not relieved by rest

2 Limitation of lumbar spine motion in sagittal and frontal planes

3 Chest expansion decreased relative to normal values for age and sex

Radiographic Criteria

4 Bilateral sacroiliitis, grade 2-4

5 Unilateral sacroiliitis, grade 3-4

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al disease; and (6) no medical

con-traindications to the initiation of

treatment.21Braun et al22summarized

the results of anti–TNF-α medications

in more than 300 patients enrolled in

clinical trials, which indicated

signif-icant (P < 0.0001) short-term

improve-ment in disease activity, function, and

quality of life Long-term efficacy of

these medications is being studied in

ongoing clinical trials Early medical

intervention may improve patient

function, lessen pain, and delay

dis-ease progression

The physician is responsible for

educating the patient about the

dis-ease and its natural history An

activ-ity program of physical exercise,

in-cluding stretching and postural

alignment, should be consistently

fol-lowed A medium-firm mattress is

recommended, and the patient may

benefit from sleeping supine with one

pillow to maintain a more normal

sagittal spinal alignment and prevent

hip flexion contractures Ultimate

ankylosis of the spine in an anatomic

position, an achievable outcome for

patients who receive early diagnosis

and counseling, is far preferable to

progressive kyphosis

Surgical Management

Indications for surgical treatment of

patients with AS are to lessen pain

and to improve function

Addition-ally, the psychosocial effects of severe

deformity are devastating, and

cor-rection may help the patient recover

vital social skills and possibly accept

having the disease THA is the most

common surgical intervention in AS

patients, followed by spinal

osteoto-mies for correction of cervical

defor-mity and thoracolumbar sagittal

bal-ance Few patients require surgical

intervention for correction of

periph-eral joint abnormalities

Hip Deformity

Thirty percent to 50% of patients

with AS exhibit involvement of the

hips; of those, 90% present

bilateral-ly.23 Hip involvement ranges from flexion contractures to complete ankylosis, often in a disabling flexed position THA consistently improves both range of motion (ROM) and function; it also decreases pain in pa-tients with AS

Patients with AS onset at an early age are more likely to require THA

Sweeney et al24reported the mean age

of AS onset in patients undergoing THA to be 19.5 years, an average of 3.5 years younger than matched con-trols with AS who had not undergone THA In addition, 16% of AS patients with onset before age 16 years re-quired THA within 20 years

Indications for THA are correction

of hip flexion contractures and reduc-tion of pain caused by degenerareduc-tion, with the goal of improved function

Patients with AS typically undergo THA at a younger age than do pa-tients having THA for osteoarthritis

THA implant survival in AS patients

is similar to that in other young pa-tients with THA It was previously thought that patients with AS were

at a significantly greater risk of de-veloping disabling heterotopic ossi-fication (HO) than other patients un-dergoing THA; however, careful review of the literature reveals that there may not be dramatically

high-er rates of HO for age- and sex-matched counterparts.25,26

Correction of hip flexion contrac-ture with THAcan restore sagittal bal-ance in patients with AS Tang and Chiu27noted that, because of the pres-ence of relative hyperextension of the hips after THA, AS patients are more prone to anterior dislocation when ac-etabular components are placed in their normal position relative to the pelvis When positioning the acetab-ular component in a patient with AS, one must account for the relationship

of the pelvis to the lumbar spine in the sagittal plane in order to avoid an excessively hyperextended hip once the patient resumes an upright posi-tion In patients with severe spinal

de-formity, corrective spinal osteotomies should be performed before THA to prevent future acetabular component malposition However, many sur-geons still concur with Lee,28who as early as 1963 stated that THA should

be performed before considering spi-nal osteotomy because improvement

in hip ROM and pain relief may ob-viate the need for spinal osteotomy

in patients with severe hip flexion de-formity Determining surgical se-quence should be based on patient circumstances

Several studies indicate that THA

is beneficial in AS patients (Table 2) The studies listed in Table 2

evaluat-ed a total of 524 THAs performevaluat-ed in

320 patients with AS Ambulatory sta-tus improved in all patients Sixty per-cent to 97% of patients reported no pain after THA.23,25,27,29-31Fifteen-year survival (ie, time to THA failure) ranged from 66% to 81.4%.23,27,31 Av-erage age at time of surgery was 38 years.23,25,27,29-31 Thirty patients had Brooker33 class 3 or 4 HO.27,29,32 Eighty-six revision THAs were performed.23,27,29-32Sweeney et al24 re-viewed charts and questionnaires of

340 patients with AS who were

treat-ed with THA Survival rates of pri-mary THA were 90%, 78%, and 64%

at 10, 15, and 20 years, respectively Survival rates of revisions were 73%, 55%, and 55% at 10, 15, and 20 years, respectively These THA survival rates and those listed in Table 2 are not dissimilar to survival rates in other young patients requiring THA.34-40

The majority of revisions in the studies listed in Table 2 were per-formed for aseptic loosening of both the femoral and acetabular compo-nents.27,30Tang and Chiu27used both uncemented and cemented implants

in their series of THA in patients with AS; they reported loosening rates of 5% and 28% in uncemented and ce-mented implants, respectively The short duration of follow-up in pa-tients receiving uncemented implants prevented Tang and Chiu from fully

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advocating their use However, more

recent preliminary clinical data39,40

support the use of uncemented

ace-tabular and femoral components in

young, active patients, including

those with AS

The level of risk for heterotopic

bone formation after THA in

pa-tients with AS remains

controver-sial Previous reports have resulted

in the impression that the rates of

postoperative HO are unacceptably

high after THA in AS patients

How-ever, careful review of the

previ-ously reported high rates of HO

af-ter THA in patients with AS shows

rates not unlike those seen in other

young patients undergoing THA

Additionally, patients with

preoper-ative ankylosis often experience

sig-nificant improvement in function

af-ter THA.23,30,41The proportion of AS

patients with functional ankylosis

secondary to HO following THA is

likely substantially lower than

pre-viously thought Patients with class

3 HO, nevertheless, experienced

sig-nificant (P < 0.01) improvement in

ROM, pain relief, and independent ambulation after THA.41 Higher rates of HO after THA have been re-ported in AS patients who have un-dergone repeat operations, who have experienced postoperative in-fection, who were treated with a transtrochanteric approach, and/or who had an active disease (reflected

by elevated ESR or CRP level)

Brinker et al25found similar rates of

HO in 12 patients with AS and in an age-matched control group without

AS who underwent THA High-risk patients with preoperative ankylo-sis, with previous hip surgery, with previous infection, and/or in whom

a transtrochanteric approach to the hip was used should be considered for HO prophylaxis with indo-methacin or low-dose radiation The benefits of HO prophylaxis in other patients with AS has not been defin-itively determined

Other Peripheral Joint Involvement

The rate of shoulder and knee in-volvement in AS ranges from 25% to 70%.42Although little has been

report-ed on managing the shoulder and knee in patients with AS, Finsterbush

et al32noted marked improvement in walking, function, and pain in 21 of

23 patients with severe hip, knee, and ankle involvement who were treated with surgery and rehabilitation They did not address survivorship or the treatment of knee or ankle involve-ment Parvizi et al43recently

report-ed on 30 total knee arthroplasties in

20 patients (mean age, 55 years) who were followed for a mean of 11.2 years Knee Society pain scores

im-proved significantly (P < 0.0001), from

a mean of 14 preoperatively to a mean

of 76.3 at final follow-up Knee Soci-ety function scores improved from 16.3 to 58.7 There was essentially no improvement in ROM (84.8° to 86.7°)

In both series,32,43severe preoperative

Table 2

Review of Total Hip Arthroplasty in Patients With Ankylosing Spondylitis

Study

Patients/

Hips (N)

Ce-mented/

Unce-mented

Preop-erative Anky-losis (N)

Mean Age at Surgery (yrs)

Mean Follow-up (yrs [range])

HO:

Brooker Class 1-2/3-4

Mean Survival at 10/15/20

yr (%)

Revi-sions (N)

Patients With-out Pain (%)

Ambu-lation Status

Finster-bush et

al32

Shih

Brinker

Sochart

and

Porter30

(1

mo-30 yr)

Tang

and

Chiu27

49 U

(2-28)

Lehti-mäki

et al31

Joshi

— = no data, C = cemented, HO = heterotopic ossification, I = improved, U = uncemented

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morbidity justified total knee

arthro-plasty as a reliable means of

provid-ing durable pain relief and modest

improvement in function, regardless

of the high incidence of postoperative

stiffness

In their review of 158 patients with

AS, Emery et al44identified 52 with

shoulder involvement, 6 of whom

had acute inflammatory arthropathy

of the sternoclavicular or

acromio-clavicular joints Most symptoms

re-sponded to NSAIDs Forty-one of the

52 patients with shoulder

involve-ment experienced restriction of

scapulothoracic motion, which was

always bilateral; of these, 34 patients

had involvement of the

scapulotho-racic joint with loss of ROM (forward

flexion, abduction, and external

rota-tion), and 7 patients had asymmetric

scapulothoracic and glenohumeral

involvement Most shoulder

involve-ment was minimally disabling and

re-sponded to nonsurgical management

Destructive glenohumeral

involve-ment, although rare, was the most

disabling The presence of limited

scapulothoracic ROM may

compli-cate treatment of AS patients, similar

to the difficulties encountered during

treatment of other patients with

de-structive glenohumeral arthropathy

Spine

In AS, chronic inflammation of the

zygapophyseal joints leads to

pro-gressive fusion of the spine,

typical-ly in a caudal to cranial fashion As

spinal segments become

progressive-ly fused, pain often disappears

Os-teopenia is common in patients with

AS Rigid fused sections of the spine

concentrate and convert compressive

stresses to shear stresses at the

atlanto-occipital, cervicothoracic, and

thoracolumbar junctions This can

lead to fracture, pseudarthrosis, and

spondylodiscitis Additionally,

pro-gressive increases in cervical and

tho-racic kyphosis, combined with loss of

lumbar lordosis and of motion at the

hip joints, can result in loss of

sagit-tal balance The resultant stooped

posture can lead to significant func-tional deficits and a deformed ap-pearance Incapacitating symptoms include the inability to lie in bed or

to look straight ahead Patients with cervical ankylosis may have

difficul-ty with horizontal gaze even after correction of lumbar alignment Sur-gical intervention also may be indi-cated for management of deformity, pseudarthrosis, fracture, and spondy-lodiscitis

Cervical Deformity

Correction of deformity at the

lev-el of the cervical spine is indicated for patients in whom sagittal balance has been maintained or regained

surgical-ly, but whose kyphotic chin-brow an-gle has significantly impaired the vi-sual field or interfered with either hygiene or swallowing These pa-tients may exhibit chin-on-chest de-formity Patients with evidence of fracture, hypermobility of the cervi-cal spine, and subluxation of C1-C2 leading to kyphotic deformity should

be treated with gradual halo traction

to restore accurate alignment of the spinal canal and chin-brow angle Pa-tients with C1-C2 instability should

be treated initially with cervical trac-tion in a halo, followed by posterior arthrodesis Osteotomy of the cervi-cal spine should be limited to patients with rigid deformities Evidence of re-cent fracture, characterized by an overt fracture or by new onset of pain

in the cervical spine of a person pre-viously without pain, is a contrain-dication to osteotomy

Urist45first described extension os-teotomy of the cervical spine for cor-rection of cervical deformity This operation is performed under local anesthesia with the patient awake to facilitate neurologic monitoring dur-ing the reduction The level of correc-tion is centered at C7-T1 because the canal is relatively wide at this level and the cervical cord and nerve roots are relatively mobile In addition, the vertebral vessels do not enter the lat-eral masses at this level In 1998, Liew

and Simmons46recommended resec-tion extension osteotomy followed by posterior fusion and immobilization, with either halo or collar postopera-tively, depending on the fixation method In 1997, McMaster47

report-ed the results of extension osteotomy

at the C7-T1 level in AS patients with severe flexion deformity of the cervi-cal spine All 15 patients (mean age,

48 years) had a mean correction of 54° One patient became

quadriparet-ic, two had unilateral C8 palsies, and four experienced subluxations Neu-rologic problems were attributed to dural traction (ectasia), posterior el-ement impingel-ement of C8 roots, and subluxation Rigid fixation using plates and rod-and-screw constructs may decrease neurologic complica-tions by maintaining stability during and after osteotomy

Thoracolumbar Deformity

Corrective osteotomy of the lum-bar spine is indicated to correct sag-ittal balance when correction of hip flexion contractures does not restore sagittal balance and the patient is markedly disabled For patients with pseudarthrosis, Simmons48

advocat-ed correction of kyphotic deformity

by spine osteotomy to convert trans-lational forces to compressive forces

at the site of pseudarthrosis Van Royen et al15devised an equa-tion correlating sagittal balance and physiological end plate angle Nomo-grams constructed from this informa-tion allow the surgeon to correlate the correction angle, horizontal position

of the C7 plumb line, and the level

of the spinal osteotomy, predicting the optimum level and degree of correc-tion

Most authors advocate general an-esthesia and fiberoptic intubation for thoracic and lumbar deformity cor-rection In a review of 66 spinal os-teotomies, Bridwell et al49stressed the importance of the “wake-up test” for intraoperative patient evaluation Five neurological sequelae were missed by somatosensory evoked

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po-tentials but were detected by the

wake-up test

The osteotomies can be performed

in either the lateral decubitus or prone

position Initially plaster

thoracolum-bosacral orthosis with one leg

includ-ed was usinclud-ed for postoperative

immo-bilization Since the advent of

segmental pedicle screw fixation,

many surgeons immobilize patients

in custom rigid thoracolumbosacral

orthoses with a hip extension or

fore-go postoperative immobilization

al-together Recommended periods of

immobilization vary from 2 to 4

months for closing wedge

osteoto-mies and 6 to 15 months for opening

wedge and polysegmental

osteoto-mies

The opening wedge,

polysegmen-tal wedge, and closing wedge

osteot-omies (Fig 3) are the three surgical

techniques used for correcting

lum-bar kyphosis In 1945, Smith-Petersen

et al50 first described an opening

wedge osteotomy for correction of

lumbar kyphosis in six patients

Hehne et al51advocated

polysegmen-tal wedge osteotomy (Fig 4), which

was first described by Wilson and

Turkell52 in 1949 In 1985,

Thomas-en53subsequently described a

mono-segmental closing wedge osteotomy

of the lumbar spine, in which the

pos-terior elements, pedicles, and a

por-tion of the vertebral body are

resect-ed to accomplish the correction (the

so-called pedicle subtraction

osteot-omy)

In their meta-analysis of articles published between 1945 and 1998, Van Royen and De Gast14reported on

856 patients with AS who underwent correction of fixed kyphotic thora-columbar deformity There were 451 opening wedge osteotomies, 249 polysegmental wedge osteotomies, and 156 closing wedge osteotomies

Mean patient age was 41 years at the time of operation; mean correction

obtained was 40.3° for opening wedge osteotomies, 40.3° for polyseg-mental wedge osteotomies, and 36.5° for closing wedge osteotomies Of the

856 patients who underwent surgery,

34 (4%) died from intraoperative or postoperative complications The au-thors noted a tendency toward less severe complications with closing wedge osteotomy Jaffray et al54 re-ported that closing wedge osteotomy

Figure 4 Preoperative (A) and postoperative (B) weight-bearing lateral radiographs of a

41-year-old man who underwent polysegmental closing wedge osteotomy of the lumbar spine for correction of severe kyphotic deformity and loss of sagittal balance Lumbar lordosis was corrected from 37° preoperatively to 60° postoperatively, which resulted in a 16-cm correc-tion of the patient’s sagittal balance to neutral.

Figure 3 Bone removed for opening wedge osteotomy (A), polysegmental closing wedge osteotomy (B), and closing wedge osteotomy (C).

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can be performed even when the

an-terior longitudinal ligament is

com-pletely ossified The anterior column

is not forcibly extended in this

tech-nique, thereby minimizing the risk of

abdominal aortic injury In their

se-ries of 45 patients undergoing

pedi-cle subtraction closing wedge

osteot-omy, Kim et al55 demonstrated an

ability to effect sagittal and coronal

balance correction with one- and

two-level closing wedge osteotomies with

no long-term neurologic sequelae

Berven et al56also reported correction

of sagittal balance with closing wedge

osteotomy Pedicle subtraction

osteot-omy avoids intraoperative nerve root

compression resulting from closing

down the neural foramina as the

spine is extended

Halm et al57collected functional

outcome data from 148 of 175 patients

who had spinal deformity correction

surgery between 1979 and 1988

Im-provement was noted in 47 of the 60

items of the modified Arthritis Impact

Measurement Scales at a mean

follow-up of 4.8 years Eighty-eight

percent of patients were very satisfied

with the results of surgery, and 60.9%

were able to return to work In a

se-ries of 83 patients (5 with AS)

under-going spinal osteotomy to correct

fixed sagittal and/or coronal plane

deformity, Ahn et al58demonstrated

a statistically significant correlation

between patient functional outcome

and satisfaction with sagittal

lordo-sis >25° (P = 0.034) and coronal plumb

alignment <2.5 cm (P = 0.041) Suk et

al59 prospectively evaluated 34

pa-tients with AS after they underwent

pedicle subtraction extension

osteot-omy for correction of sagittal plane

deformity Correction of chin-brow

angle from−10° to 10° resulted in

sig-nificantly (P = 0.000) better

horizon-tal gaze Additionally, the functional

scores of items related to the

horizon-tal gaze were higher than the overall

mean score Because of the neck

stiff-ness of AS patients, correcting the

chin-brow angle is better for them

than absolute correction of coronal

and sagittal balance would be, be-cause correcting the chin-brow angle improves these patients’ visual field

The importance of planning

deformi-ty correction so as to correct visual fields is essential in AS patients

It is unclear which type of spinal osteotomy is most appropriate for the treatment of severe kyphotic

deformi-ty, although closing wedge osteoto-mies appear to have a lower incidence

of complications Deformity correc-tion surgery in AS patients is associ-ated with dural tears secondary to du-ral ectasia, transient nerve root compression, loss of correction, im-plant failure, postoperative spinal in-stability, and aortic injury

Fortunate-ly, the number of patients with severe kyphotic deformity is decreasing be-cause of improved medical manage-ment and earlier detection.60

Fracture

Generalized spinal osteopenia is common in patients with AS Spinal fracture often is caused by minor trauma and can be missed on spine radiographs because of distortion of the normal spinal anatomy by

ectop-ic bone formation, erosions, sclerosis, disk ossification, vertebral wedging,

or difficulty with patient positioning

Onset of pain after even minor

trau-ma should suggest a fracture, which may not be visible on plain radio-graphs.61Fractures frequently occur

at the C7-T1 junction and are difficult

to visualize radiographically Bone scan, MRI, or fine-cut CT can be used

to evaluate AS patients who present with cervical pain (Fig 5)

The rigid, unsupported, osteo-porotic cervical spine is most suscep-tible to hyperextension injuries, and the cervical spine is the most common site of fracture in patients with AS The drastic increase in stiffness at the cer-vicothoracic junction, combined with the lever arm of the fused cervical spine and weight of the head, makes fractures at the C6-C7 and C7-T1 lev-els most common The lumbar and thoracic spines are more resistant to

fracture because the anterior and pos-terior longitudinal ligaments are more thoroughly ossified than in the cer-vical spine Spinal fractures

typical-ly occur through the ossified disk and vertebral body; vertebral body frac-tures are more common and have a higher risk of neural compromise, partly as a consequence of a higher incidence of epidural hematoma and the possibility of the newly mobile fracture severely translating, result-ing in neural injury.61

AS patients who sustain spinal frac-tures typically have high rates of neu-rologic injury.62Missed fractures are frequent and often present as rapidly progressing cervical kyphosis Surgi-cal treatment is more commonly used, especially in patients with neurologic compromise, obscured visual fields, pseudarthrosis, or recurrent fracture When traction or internal fixation are

Figure 5 T2-weighted sagittal reconstruction magnetic resonance image of a 46-year-old man with a C7-T1 disk space fracture with subluxation and cord compression Fast spin-echo sequences may show edema, thereby al-lowing fracture localization, even without fracture displacement.

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used to manage these injuries, the neck

should be aligned to prefracture

position, not necessarily to a normal

position (Fig 6) Minor findings in

pa-tients with AS may be associated with

substantial instability in the cervical

spine, secondary to the altered

bio-mechanics of the fused spine in

ad-dition to osteopenia and the

concen-tration of forces at the cervico-occipital

and cervicothoracic junctions

Preoperative evaluation of the

cervical spine is essential when

ma-nipulating the neck during

intuba-tion and patient posiintuba-tioning

Physi-cians also must be aware that,

because the atlanto-occipital joint is

last to fuse, atlantoaxial instability

may occur Instability is usually

demonstrated on lateral

flexion-ex-tension views of the neck, where the

atlantodens and posterior

atlan-todens intervals are measured An

atlantodens interval >3.5 mm is

in-dicative of instability A difference of

7 mm indicates disruption of the

alar ligaments, and a difference >9 to

10 mm or a posterior atlantodens

in-terval >14 mm is associated with an

increased risk of neurologic injury

and usually requires surgical inter-vention

Spondylodiscitis

Spondylodiscitis has been

report-ed to occur in 5% to 23% of patients with AS; 50% of these patients are asymptomatic.63,64Spondylodiscitis, which presents as an erosive

sclerot-ic process, characteristsclerot-ically involves the intervertebral disk and adjacent bone It can be confused radiograph-ically with diskitis and pseudarthro-sis Unlike diskitis, biopsies of these lesions demonstrate chronic inflam-matory changes with no infective or-ganisms.63,64Spondylodiscitis lesions occur at the level of the intervertebral disk rather than through the vertebral body, as in pseudarthrosis Pseudar-throsis often presents as new back pain in a previously asymptomatic in-dividual and often arises after minor trauma Most cases of spondylodis-citis and pseudarthrosis resolve with nonsurgical management Surgical stabilization may be required in pa-tients with intractable pain,

associat-ed posterior element disruption, and fracture It is important to assess

ky-photic deformity before stabilization because deformity can create shear stresses at the site of spondylodisci-tis or pseudarthrosis These transla-tional forces can be converted to com-pressive forces at the time of stabilization, leading to higher rates

of fusion

Summary

AS, which affects approximately 350,000 people in North America and 600,000 in Europe (worldwide prevalence, 0.9%), belongs to a larger group of spondyloarthropa-thies and is characterized predomi-nantly by involvement of the sa-croiliac joints in young men who present with inflammatory low back pain in their third or fourth decade The disease primarily affects the ax-ial skeleton and is characterized by enthesopathy, which leads to fusion

of the sacroiliac joints, hips, and spine Young men presenting with inflammatory back pain and evi-dence of sacroiliitis should be screened for AS; most patients are HLA-B27–positive

Management of AS remains pal-liative, with NSAIDs continuing to

be the mainstay of treatment How-ever, new anti–TNF-α drugs offer the promise of arresting the progres-sion of AS AS patients presenting with back or neck pain after minor trauma should be considered to have spinal fractures until defini-tively proven otherwise with CT or MRI THA can significantly improve function, pain, and deformity in AS patients with severe hip involve-ment Results are comparable to those in other young patients with severe hip pathology Because the incidence of HO in AS patients undergoing THA may not be sig-nificantly more than that of similar non-AS patients undergoing THA, the routine use of HO prophylaxis in

AS patients should be reserved for

Figure 6 Preoperative (A) and postoperative (B) lateral radiographs of a 31-year-old man

who sustained a C5-C6 fracture through the disk space Posterolateral fusion was done with

lateral mass plates and posterior element wiring.

Ngày đăng: 12/08/2014, 03:21

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