Chapter 134 Botulism (Part 2) pot
Chapter 134. Botulism (Part 2) pot
... dietary exposure (i.e., features suggesting
an aerosol attack)
Chapter 134. Botulism
(Part 2)
Clinical Manifestations
Food-Borne Botulism
After ingestion of food containing toxin, illness ... inhalational botulism) or as a contaminant in material to be ingested
(producing food-borne botulism) . Inhalational botulism resembles food-borne
illness, but gastrointestinal s...
Chapter 021. Syncope (Part 2) potx
... syncope can be associated with
prolonged asystole and hypotension, resulting in hypoxic-ischemic injury.
Chapter 021. Syncope
(Part 2)
Disorders of Vascular Tone or Blood Volume
Disorders
Chapter 043. Jaundice (Part 2) potx
... is
impaired and the glucuronides are present in serum in increasing amounts. By
Chapter 043. Jaundice
(Part 2)
Measurement of Serum Bilirubin
The terms direct- and indirect-reacting bilirubin ... hepatic/biliary removal of the pigment.
Hyperbilirubinemia may result from (1) overproduction of bilirubin; (2) impaired
uptake, conjugation, or excretion of bilirubin; or (3) regurgi...
Chapter 134. Botulism (Part 1) pdf
... Chapter 134. Botulism
(Part 1)
Harrison's Internal Medicine > Chapter 134. Botulism
Definition
Botulism is a paralytic disease caused by potent protein neurotoxins ... (Fig.
134- 1). Cure follows sprouting of new nerve terminals.
Figure 134- 1
involvement and proceeds caudally to involve the extremities. Cases may be
classified as (1) food-borne botulism, ....
Chapter 134. Botulism (Part 4) ppsx
... failure and long
term outcome in black tar heroin–
associated wound botulism. Chest 120:562,
Chapter 134. Botulism
(Part 4)
Botulism: Treatment
Patients should be hospitalized and monitored ... Prevention: Botulism in the United States,
1899-
1996: Handbook for epidemiologists, clinicians, and laboratory workers.
Atlanta, CDC, 1998 (http://www.bt.cdc.gov/agent /botulism/ )...
Chapter 012. Pain: Pathophysiology and Management (Part 2) pot
... messengers of threats to tissue injury but also play an active role in
Chapter 012. Pain:
Pathophysiology and Management
(Part 2)
Sensitization
When intense, repeated, or prolonged stimuli are ... activated (Fig. 12 -2). An example is substance P, an 11-
amino-acid peptide. Substance P is released from primary afferent nociceptors and
has multiple biologic activities. It is a po...
Chapter 018. Fever and Rash (Part 2) pot
... Chapter 018. Fever and Rash
(Part 2)
Centrally Distributed Maculopapular Eruptions
Centrally distributed rashes, ... confused
with Fordyce's spots (ectopic sebaceous glands), which have no erythematous
halos and are found in the mouth of healthy individuals. Koplik's spots may briefly
overlap with ... begins as discrete erythematous lesions, which become confluent
as...
Chapter 020. Hypothermia and Frostbite (Part 2) pot
... initial stimulation by hypothermia, there is progressive depression of
all organ systems. The timing of the appearance of these clinical manifestations
varies widely (Table 20 -2). Without knowing ... patient with
mild hypothermia should raise suspicion of an overdose or CNS dysfunction due
to infection or trauma.
Table 20-2 Physiologic Changes Associated with Accidental
Hypothermia
nt...
Chapter 023. Weakness and Paralysis (Part 2) pot
... motor neurons have their cell bodies in layer V of the primary motor cortex
Chapter 023. Weakness and Paralysis
(Part 2)
Pathogenesis
Upper Motor Neuron Weakness
This pattern of weakness
Chapter 026. Confusion and Delirium (Part 2) potx
...
Chapter 026. Confusion and Delirium
(Part 2)
Epidemiology
Delirium is a common disease, but its reported incidence